NMN

Based on the biological mechanisms involved, the short answer is yes, but it is more accurate to say that destroying free radicals preserves your existing levels rather than "creating" new  mean NMN (Nicotinamide Mononucleotide) rather than MNM. NMN is the direct precursor to NAD+ (Nicotinamide Adenine Dinucleotide), the critical molecule for energy and cellular repair.

Here is the breakdown of why destroying free radicals results in higher natural levels of NMN and NAD+.

1. The "Bathtub" Analogy

Think of your body's NAD+ levels like water in a bathtub.

 * ** The Faucet (Production):** Your body naturally makes NAD+ using precursors like NMN.

 * The Drain (Consumption): Your body uses up NAD+ to fight stress, repair DNA, and maintain immunity.

Free radicals are like a massive hole in the drain. When you have high oxidative stress (too many free radicals), your body is forced to pour all its NAD+ down the drain to fix the damage. By "destroying" free radicals (using antioxidants), you plug that hole. You aren't necessarily turning up the faucet, but because the drain is stopped, the water level (NAD+) naturally rises.

2. The Mechanism: How Free Radicals Steal NAD+

There are two main enzymes that eat up your NAD+ when free radicals are present:

 * PARPs (The DNA Repairmen): Free radicals are unstable molecules that smash into your DNA, causing breaks. When your DNA is damaged, enzymes called PARPs (Poly-ADP-ribose polymerases) rush in to fix it. PARPs are "expensive" to run—they consume huge amounts of NAD+ fuel to perform these repairs. If you have fewer free radicals, you have less DNA damage, PARPs stay dormant, and your NAD+ is spared.

 * CD38 (The Inflammation Enzyme): Chronic oxidative stress leads to inflammation. Inflammation triggers an enzyme called CD38, which is notorious for chewing up NAD+ without necessarily giving much benefit in return. Lowering oxidative stress quiets this enzyme, further preserving your supply.

3. The Result: A Positive Cycle

When you reduce free radicals (through diet, fasting, or antioxidants like Glutathione or Vitamin C), you stop the "emergency use" of NAD+. This allows your body to use that NAD+ for its "thriving" functions instead, such as:

 * Sirtuins: These are the "longevity genes" that only work when NAD+ is available. They protect against aging and improve metabolism.

 * Energy Production: More NAD+ means better mitochondrial function (more ATP energy).

Summary

If you destroy free radicals, you stop the biological "leak." This allows your natural NMN and NAD+ levels to remain higher naturally because they aren't being exhausted trying to repair the damage that free radicals would have caused.

Free Radicals and Seizures

 There is a very strong, well-documented link between free radicals and seizures.

In fact, neuroscientists often describe this relationship as a "vicious cycle." Free radicals can trigger seizures, and seizures, in turn, create a massive explosion of new free radicals, which makes future seizures more likely.

Here is how that mechanism works, keeping in mind the NAD+ context we just discussed.

1. The "Rusting Wire" Analogy

Think of your brain's neurons as electrical wires coated in insulation.

 * The Insulation (Cell Membranes): Your brain cells are wrapped in fatty membranes that help conduct electricity properly.

 * The Rust (Free Radicals): Free radicals love to attack fats (a process called lipid peroxidation).

When free radicals "eat" the fatty coating of your neurons, the "insulation" wears down. This causes the electrical signals to leak or misfire. If enough wires cross or short-circuit due to this damage, the result is a sudden electrical storm—a seizure.

2. The Energy Failure (The NAD+ Connection)

This is where your interest in NAD+ becomes very relevant.

To stop a seizure, your brain needs massive amounts of energy to pump ions back into place and "calm down" the electrical storm.

 * The Problem: As we discussed, free radicals drain your NAD+.

 * The Result: Without enough NAD+, your mitochondria (power plants) cannot produce enough ATP energy to regulate the brain's electricity. The brain becomes "hyperexcitable" because it lacks the energy to calm itself down.

(Next blog what is ATP)

3. The Glutamate Loop

There is a specific neurotransmitter involved here called Glutamate.

 * Glutamate is the "Go" signal in your brain (it excites neurons).

 * Normally, your brain quickly vacuums up extra Glutamate to prevent over-excitement.

 * Free radicals damage the vacuums. When oxidative stress is high, the transporter proteins responsible for clearing Glutamate stop working. Glutamate builds up, over-stimulating the neurons until they snap into a seizure.

Summary

Free radicals lower your "Seizure Threshold."

Everyone has a threshold—a point where, if stress gets high enough, a seizure occurs. High levels of free radicals lower this ceiling, meaning it takes much less stress or stimulation to trigger a seizure than it would in a healthy brain.

In short: Reducing free radicals (and boosting NAD+) helps "insulate" the wires and provides the energy the brain needs to prevent these electrical storms.

What is ATP (Your Battery) How to stay charged!

ATP 

(Adenosine Triphosphate) is essentially your body’s "fully charged battery."

Every single cell in your body needs electricity to function—whether it’s a muscle contracting, a neuron firing, or your heart beating. ATP is that electricity.

1. What is it exactly?

Chemically, it is a molecule of Adenosine bonded to a tail of 3 Phosphate groups (hence "Tri"-phosphate).

Think of those three phosphates like a compressed spring. They are held together by high-energy bonds. When your body needs energy, it snaps off the last phosphate.

 * The Snap: This releases a burst of energy.

 * The Result: You are left with ADP (Adenosine Diphosphate), which is now a "dead battery."

Your body’s entire goal is to constantly find energy (from food) to re-attach that third phosphate and "recharge" the battery back into ATP.

2. How does the body get it?

Your body manufactures ATP in a massive factory process called Cellular Respiration. It uses the food you eat (glucose, fats, proteins) and oxygen to recharge those dead batteries.

There are three main stages, and this connects directly to your interest in NAD+:

Stage 1: Glycolysis (The "Quick & Dirty" Method)

 * Location: Outside the mitochondria (in the cell fluid).

 * Process: Splits glucose in half.

 * Yield: Very small amount of ATP (2 molecules).

 * Note: This is fast but inefficient. It doesn't need oxygen.

Stage 2: The Krebs Cycle (The "Prep Work")

 * Location: Inside the Mitochondria.

 * Process: It strips electrons from the broken-down food.

 * The Hero: This is where NAD+ shines. NAD+ picks up these high-energy electrons (becoming NADH) and acts like a shuttle bus, ferrying them to the next stage.

Stage 3: The Electron Transport Chain (The "Big Payoff")

 * Location: The inner membrane of the Mitochondria.

 * Process: The "shuttle buses" (NADH) drop off the electrons here. These electrons power a massive turbine (ATP Synthase).

 * Yield: Huge amount of ATP (30-34 molecules).

 * Requirement: This stage requires oxygen. (This is why you breathe; oxygen is the final bucket that catches the used electrons).

The "NAD+ Connection"

To tie this back to your previous questions:

 * No NAD+ = No Energy: If you lack NAD+, you have no "shuttle buses" to carry the energy from your food to the turbine. The factory shuts down.

 * Free Radicals: If free radicals damage the mitochondria (the factory), you can't run Stage 3 effectively. You are stuck using only Stage 1 (Glycolysis), which produces very little energy and creates acidic byproducts (lactate).

Summary: You eat food to get raw materials, you breathe to keep the factory running, and your mitochondria use NAD+ to turn those materials into ATP—the energy that powers your life.

But the Ion needs to be Charged.

See next blog 

Sources For ATP

Here are the sources backing up the mechanisms we discussed. I have formatted them as direct, copyable links so you can easily save or explore them.

1. Free Radicals & Seizures (The "Rusting Wire" & Vicious Cycle)

This review explains the "vicious cycle" where seizures produce free radicals, and free radicals (via oxidative stress) damage neurons to cause more seizures.

 * Source: Oxidative Stress and Epilepsy: Literature Review (National Institutes of Health)

 * Link: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3403512/

2. The Glutamate Connection (The "Vacuum" Failure)

This study details how oxidative stress damages the transporters (the "vacuums") responsible for clearing glutamate, leading to the hyperexcitability that triggers seizures.

 * Source: Mitochondrial Oxidative Stress in Temporal Lobe Epilepsy (National Institutes of Health)

 * Link: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3236664/

3. How Free Radicals Deplete NAD+ (The "Drain")

This paper provides the deep dive into how DNA damage (caused by free radicals) triggers PARP enzymes, which then rapidly consume NAD+ stores, leading to cell death or dysfunction.

 * Source: NAD+ Depletion Is Necessary and Sufficient for PARP-1-Mediated Neuronal Death (Journal of Neuroscience)

 * Link: https://www.jneurosci.org/content/30/8/2967

4. NAD+ and ATP Production (The "Factory")

A comprehensive overview of how NAD+ acts as the critical "shuttle bus" for electrons in the mitochondria to produce ATP, and how this relates to metabolic health.

 * Source: Role of NAD+ in Regulating Cellular and Metabolic Signaling Pathways (National Institutes of Health)

 * Link: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7973386/